lv trabeculation involving apex and inferolateral wall and false cord | left ventricular trabeculation triad lv trabeculation involving apex and inferolateral wall and false cord This report summarizes the evidence concerning the etiology, pathophysiology, and clinical relevance of excessive trabeculation. Gaps in current knowledge of the clinical relevance of .
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0 · trabeculation in the heart
1 · pathophysiology of trabeculation
2 · lv trabeculation in athletes
3 · lower ventricular trabeculation
4 · left ventricular trabeculation triad
5 · left ventricular trabeculation
6 · left ventricle trabecular
7 · excessive trabeculation of left ventricle
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trabeculation in the heart
This report summarizes the evidence concerning the etiology, pathophysiology, and clinical relevance of excessive trabeculation. Gaps in current knowledge of the clinical relevance of .Echocardiography at preparticipation screening documented the presence of mildly reduced left ventricular systolic function, mild eccentric aortic regurgitation, and a severely dilated LV with . Patients with LVNC frequently (75%) express symptoms of LV dysfunction whereas athletes are asymptomatic. These individuals also frequently (66%) demonstrate a .
An echocardiogram showed an ejection fraction of 34% with normal valvular function. Prominent trabeculations were noted in the lateral and inferior walls, with hypokinesis in mid-ventricular .
If the phenotype is present in a patient with high pretest probability based on family history, symptoms, or imaging evidence of abnormal left ventricular size or function, we .
This report summarizes the evidence concerning the etiology, pathophysiology, and clinical relevance of excessive trabeculation. Gaps in current knowledge of the clinical relevance of .The presence of excessive left ventricular (LV) trabeculation remains enigmatic: both seen on imaging in congenital, inherited, and acquired cardiomyopathies and in normal, healthy . The left ventricle (LV) is affected by many diseases with different clinical and morphological features. Within this broad spectrum, a subset of heterogeneous diseases is .
Cardiomyopathies with excessive trabeculations, often labelled as ‘left ventricular non-compaction’, can lead to complications, including heart failure, life-threatening heart rhythm .
Left ventricular hypertrabeculation (LVHT) is a phenotype with multiple etiologies and variable clinical presentation and significance. It is characterized by a 2-layer myocardium .This report summarizes the evidence concerning the etiology, pathophysiology, and clinical relevance of excessive trabeculation. Gaps in current knowledge of the clinical relevance of .
Echocardiography at preparticipation screening documented the presence of mildly reduced left ventricular systolic function, mild eccentric aortic regurgitation, and a severely dilated LV with . Patients with LVNC frequently (75%) express symptoms of LV dysfunction whereas athletes are asymptomatic. These individuals also frequently (66%) demonstrate a .An echocardiogram showed an ejection fraction of 34% with normal valvular function. Prominent trabeculations were noted in the lateral and inferior walls, with hypokinesis in mid-ventricular . If the phenotype is present in a patient with high pretest probability based on family history, symptoms, or imaging evidence of abnormal left ventricular size or function, we .
This report summarizes the evidence concerning the etiology, pathophysiology, and clinical relevance of excessive trabeculation. Gaps in current knowledge of the clinical relevance of .The presence of excessive left ventricular (LV) trabeculation remains enigmatic: both seen on imaging in congenital, inherited, and acquired cardiomyopathies and in normal, healthy .
pathophysiology of trabeculation
The left ventricle (LV) is affected by many diseases with different clinical and morphological features. Within this broad spectrum, a subset of heterogeneous diseases is .Cardiomyopathies with excessive trabeculations, often labelled as ‘left ventricular non-compaction’, can lead to complications, including heart failure, life-threatening heart rhythm . Left ventricular hypertrabeculation (LVHT) is a phenotype with multiple etiologies and variable clinical presentation and significance. It is characterized by a 2-layer myocardium .This report summarizes the evidence concerning the etiology, pathophysiology, and clinical relevance of excessive trabeculation. Gaps in current knowledge of the clinical relevance of .
Echocardiography at preparticipation screening documented the presence of mildly reduced left ventricular systolic function, mild eccentric aortic regurgitation, and a severely dilated LV with .
Patients with LVNC frequently (75%) express symptoms of LV dysfunction whereas athletes are asymptomatic. These individuals also frequently (66%) demonstrate a .An echocardiogram showed an ejection fraction of 34% with normal valvular function. Prominent trabeculations were noted in the lateral and inferior walls, with hypokinesis in mid-ventricular . If the phenotype is present in a patient with high pretest probability based on family history, symptoms, or imaging evidence of abnormal left ventricular size or function, we .This report summarizes the evidence concerning the etiology, pathophysiology, and clinical relevance of excessive trabeculation. Gaps in current knowledge of the clinical relevance of .
The presence of excessive left ventricular (LV) trabeculation remains enigmatic: both seen on imaging in congenital, inherited, and acquired cardiomyopathies and in normal, healthy . The left ventricle (LV) is affected by many diseases with different clinical and morphological features. Within this broad spectrum, a subset of heterogeneous diseases is .
Cardiomyopathies with excessive trabeculations, often labelled as ‘left ventricular non-compaction’, can lead to complications, including heart failure, life-threatening heart rhythm .
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lv trabeculation involving apex and inferolateral wall and false cord|left ventricular trabeculation triad
